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M9490727.TXT
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1994-09-24
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Document 0727
DOCN M9490727
TI Hypothesis for retroviral causation of rheumatoid arthritis.
DT 9411
AU Wilder RL; Inflammatory Joint Diseases Section, National Institute of;
Arthritis and Musculoskeletal and Skin Disease, National; Institutes of
Health, Bethesda, MD 20892.
SO Curr Opin Rheumatol. 1994 May;6(3):295-9. Unique Identifier : AIDSLINE
MED/94338863
AB Over the past year, convincing direct evidence that rheumatoid arthritis
is caused by a retrovirus has not been presented. Strong support for
this hypothesis, however, comes from an impressive body of recent work
demonstrating that human T-cell lymphotropic virus type I causes a
destructive arthropathy that has many of the same features as rheumatoid
arthritis. Additional strong support for the hypothesis is provided by
the resemblance of the arthropathies induced by caprine arthritis
encephalitis and the ovine maedi-visna viruses to rheumatoid arthritis.
These retroviral infections emphasize the potential complexity of
detecting retroviral involvement and proving its importance in the
causation of rheumatoid arthritis. Retroviruses clearly possess diverse
pathways for involvement in autoimmune diseases.
DE Animal Arthritis-Encephalitis Virus, Caprine/PHYSIOLOGY Arthritis,
Rheumatoid/GENETICS/*MICROBIOLOGY AIDS-Related Opportunistic Infections
Human HIV Infections/COMPLICATIONS HIV-1/PHYSIOLOGY HIV-2/PHYSIOLOGY
HTLV-I Infections/COMPLICATIONS Lentivirus Infections/COMPLICATIONS
Retroviridae/*PHYSIOLOGY Retroviridae
Infections/*COMPLICATIONS/MICROBIOLOGY RNA, Viral/GENETICS/ISOLATION &
PURIF Visna-Maedi Virus/PHYSIOLOGY JOURNAL ARTICLE REVIEW REVIEW,
TUTORIAL
SOURCE: National Library of Medicine. NOTICE: This material may be
protected by Copyright Law (Title 17, U.S.Code).